Analysis Tools
cellular
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• with prior treatment with glucocorticoid stress hormone followed by middle cerebral artery occlusion
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hematopoietic system
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• prior to glucocorticoid stress hormone treatments fail to exhibit a reduction in numbers of infiltrating monocytes in infarcted hemisphere unlike in wild-type mice
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homeostasis/metabolism
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• with prior treatment with glucocorticoid stress hormone followed by middle cerebral artery occlusion
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• prior treatment with glucocorticoid stress hormone fail to exhibit a reduction in numbers of infiltrating monocytes and IL6 protein levels in infarcted hemisphere after middle cerebral artery occlusion unlike in wild-type mice
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immune system
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• prior to glucocorticoid stress hormone treatments fail to exhibit a reduction in numbers of infiltrating monocytes in infarcted hemisphere unlike in wild-type mice
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• in the ischemic cortex after middle cerebral artery occlusion
• in the hippocampus 12, but not 72, hours after kainic acid treatment
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• prior treatment with glucocorticoid stress hormone fails to induce a decrease in IL6 levels in the ischemic cortex 24 h after middle cerebral artery occlusion
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nervous system
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• with prior treatment with glucocorticoid stress hormone followed by middle cerebral artery occlusion
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• prior treatment with glucocorticoid stress hormone fail to exhibit a reduction in numbers of infiltrating monocytes and IL6 protein levels in infarcted hemisphere after middle cerebral artery occlusion unlike in wild-type mice
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