Analysis Tools|
Allele Symbol Allele Name Allele ID |
Tg(CMV-cre)1Cgn transgene insertion 1, University of Cologne MGI:2176180 |
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| Summary |
54 genotypes
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• calcification of the fibrous capsule surrounding the muzzle vibrissae occurs at 20 weeks of age
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• only 70% of mice survive to adulthood with some mice lost after weaning
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• in most mice
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• 4-fold in most mice
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• one mouse exhibits hyperplasia of lymphoid tissue at 73 weeks
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• one mouse exhibits small intestinal adenoma at 79 weeks
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• predisposition in aged mice
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• including cataracts and watery eyes in some mice
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• facial dysmorphia
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• in some mice
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• facial dysmorphia
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• in some mice
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• in most mice
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• 4-fold in most mice
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• in some mice
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• one mouse exhibits hyperplasia of the smooth muscles of the uterus at 23 weeks
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• in some mice
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• one mouse exhibits small intestinal adenoma at 79 weeks
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• predisposition in aged mice
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• in most mice
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• 4-fold in most mice
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• predisposition in aged mice
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• in some mice
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• in some mice
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• levels are increased to 40 and 34 pg/ml in male and female mice from basal levels of less than 5 pg/ml in controls
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• mice have higher rises in serum calcium level when administered calcitrol (active form of vitamin D) than controls
• rise in serum calcium is 44% higher in males and 21% higher in females
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• bone marrow derived osteoclasts fail to dissemble actin rings in response to calcitonin
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• femur length in male mice is 4% shorter than controls at 6, 12, and 24 weeks of age
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• bone marrow derived osteoclasts fail to dissemble actin rings in response to calcitonin
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• femur length in male mice is 4% shorter than controls at 6, 12, and 24 weeks of age
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• the trabecular structures in female mice are more rod-like than plate-like
• trabecular separation in the vertebrae of male mice is increased compared to controls
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• trabecular bone formation is increased 20-40% in the femur of male mice
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• trabecular bone volume in vertebrae of female mice is reduced by 6% at 6 weeks of age
• however, female mice do not have vertebrae trabecular bone loss as the age as controls do leading to 27% increase in bone volume by 24 weeks of age
• also, connectivity density is doubled in trabecular bone
• in female femur, trabecular thickness is decreased by 11% compared to controls
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• bone marrow derived osteoclasts fail to dissemble actin rings in response to calcitonin
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• severe hemorrhage found in E12.5-E13.5 homozygotes
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• small, pale fetal liver at E12.5-E13.5
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• intercrosses of heterozygotes in the presence of a cre deleter yield no homozygous null animals at birth, showing the homozygous lethality and phenotypes of the null allele
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
Osteoarthritis-like disorder in Errfi1tm3.1Gvw/Errfi1tm3.1Gvw Tg(CMV-cre)1Cgn/0 knee joints
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• most die by 6 months of age
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• develop an osteoarthritis like phenotype in multiple synovial joints including the knee, ankle and temporal-mandibular joint
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• ankles show extensive changes including swelling, stiffness, osteophytes
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• in ankles
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• in knee joints of 3.6 months old animals
• osteophytes in knee joints of 3 months to 1 year old animals
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• develop an osteoarthritis like phenotype in multiple synovial joints including the knee, ankle and temporal-mandibular joint
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• in ankles
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• mice exhibit very-long-chain fatty acid accumulation in different lipid classes and acylcarnitines
• total C26:0 levels are increased 20-fold in brain, 44-fold in adrenals, 90-fold in testes, 14-fold in lung, 14-fold in liver and 11-fold in kidney
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• fewer than the expected number of males are obtained (6% vs the expected 25%), however time is not specified
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
| N |
• following cre recombination to remove the stop signal TSE strain ME7 incubation time is similar to that in wild-type mice
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• following cre recombination to remove the stop signal TSE strain ME7 incubation time is similar to that heterozygous Prnptm2Edin mice
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• no signs of TSE disease are seen 520 days postinfection an increased incubation time compared to wild-type or or mutants without cre recombination
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• an increased incubation time for TSE strain ME7 compared to wild-type mice or mutants without cre recombination is seen
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• embryos with high levels of Cre mediated recombination are in the process of being resorbed at E7.5, while those with more mosaic recombination survived to later ages but still die before birth
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• authors state the they observe all phenotypic features of heterozygous Ctnnb1tm1(Nfkbia)Rsu mutants in these mice, however no data is presented in J:71744
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• about 50-70% of wild-type
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• atrophy of Harderian glands
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• reduction in the number of intestinal goblet cells
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• the epithelial structure of the small intestine is loosened
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• in embryos only
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• thin fur
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• reduction in the number of intestinal goblet cells
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• in embryos only
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Mouse Models of Human Disease |
DO ID | OMIM ID(s) | Ref(s) | |
| otitis media | DOID:10754 | J:71744 | ||
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• decrease in distance traveled and vertical activity in a new environment
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• in a new environment
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• in a new environment
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• in DSS-treated mice
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• in DSS-treated mice
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• by P16
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• mice are viable and have no obvious phenotype
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• most mice die neonatally
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• in all day-of-birth mice lenses were small or absent
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• optic cups are misshapen with abnormal retinal folds and excess mesenchyme cells
• however, retinal differentiation and retinal pigment epithelium are grossly normal
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• mild kidney development defects
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• mice are smaller late gestation
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• mice are born but with rare exceptions die shortly afterwards
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• at high penetrance
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• at E18.5, mesenchyme surrounding the ureteric buds is thickened 30% and ureteric tips are dilated and misshaped
• however, nephrogenesis is normal
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• at low penetrance
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• at high penetrance
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• at high penetrance
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• at low penetrance
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• at low penetrance
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• at low penetrance
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• the frontal bone is absent
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• dysgenesis of the nasal bone is seen
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• the frontal bone is absent
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• dysgenesis of the nasal bone is seen
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• midline fusion of the sternum is incomplete
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• multiple rib fusions are seen
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• multiple vertebral fusions are seen
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• the optic placode is present at E8.75 but completely degenerated by E12.5
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• disorganized, ectopic midbrain hyperplasia of neuroectodermal precursors is seen by E12.5
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• forebrain hypoplasia is visible at E9.75
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• the cortex is disorganized
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• olfactory lobe agenesis is seen
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• the dorsal root ganglia is hyperplastic
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• dysgenesis of the nasal bone is seen
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• dysgenesis of the nasal bone is seen
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• latency to develop lung adenomas is significantly shorter compared to double mutants wild-type for Cdk4; however development of tumors characteristic of Cdk4tm1.1Bbd mutant mice is not accelerated
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• histiocytic sarcoma and other sarcomas are seen
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• anal papillomas are seen
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• focal metaplasia in the pancreatic ducts consisting of tall columnar cells with abundant apical mucin resembling gastric surface epithelial cells
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• focal metaplasia in the pancreatic ducts consisting of tall columnar cells with abundant apical mucin resembling gastric surface epithelial cells
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• latency to develop lung adenomas is significantly shorter compared to double mutants wild-type for Cdk4; however development of tumors characteristic of Cdk4tm1.1Bbd mutant mice is not accelerated
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• double mutants develop breathing difficulties after 7 - 8 months of age
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• frequent embryonic lethality; however, a significant number of double mutants survive
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• a large spectrum of multifocal lesions are seen in the lung including; small patches of bronchiolo-alveolar hyperplasias to large bronchiolo-alveolar adenomas that compress adjacent lung structures and appear to derive from type II pneumocytes
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• large bronchiolo-alveolar adenocarcinomas that compress adjacent lung structures and appear to derive from type II pneumocytes
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• histiocytic sarcoma and other sarcomas seen in 2 out of 20 and 3 out of 20 double mutants, respectively
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• anal papillomas seen in 3 out of 20 double mutants
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• MEFs expressing the oncogenic protein do not undergo proliferative senescence and proliferate continuously as immortal cells
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• a large spectrum of multifocal lesions are seen in the lung including; small patches of bronchiolo-alveolar hyperplasias to large bronchiolo-alveolar adenomas that compress adjacent lung structures and appear to derive from type II pneumocytes
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• large bronchiolo-alveolar adenocarcinomas that compress adjacent lung structures and appear to derive from type II pneumocytes
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• double mutants develop breathing difficulties after 7 - 8 months of age
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• intercrosses of heterozygotes in the presence of a cre deleter yield no homozygous null animals at birth, showing the homozygous lethality of the null allele
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
| N |
• epidermal architecture and dermal composition are normal and show no differences in efficiency of re-epithelialization, inflammatory response, granulation tissue formation, angiogenesis, and restoration of basement membrane, indicating that skin homeostasis and tissue remodeling processes are normal
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• some mice die at birth after a cross to transgenic mice expressing CMV-cre
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• some mice die within a few days of birth after a cross to transgenic mice expressing CMV-cre
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• after a cross to transgenic mice expressing CMV-cre
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• after a cross to transgenic mice expressing CMV-cre
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• after a cross to transgenic mice expressing CMV-cre
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• no viable adults are found and no postnatal lethality is detected suggesting mice die in the prenatal period
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• 83% of hearts exhibit outflow tract defects such as persistent truncus arteriosus and double outlet right ventricle
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• in 33% of E12.5 embryos
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• atrioventricular cushions fail to undergo remodeling
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• increase in apoptosis in the retina, neural crest cells, the thymus, neuroepithelium, and otic vesicles
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• decrease in proliferation in the retina, neural crest cells, the thymus, neuroepithelium, and otic vesicles
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• E13.5 or older embryos commonly exhibit craniofacial defects, including square-shaped faces, short lower jaws, cleft lip and cleft palate
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• short lower jaws 74% of embryos
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• square-shaped faces in some E13.5 or older embryos
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• in some E13.5 or older embryos
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• in some E13.5 or older embryos
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• protruding tongue due to short jaw
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• 41% of embryos exhibit defects in external ear formation
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• in some E13.5 or older embryos
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• protruding tongue due to short jaw
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• neural tube closure defects
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• square-shaped faces in some E13.5 or older embryos
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• in some E13.5 or older embryos
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• in some E13.5 or older embryos
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• protruding tongue due to short jaw
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• 41% of embryos exhibit defects in external ear formation
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• 41% of embryos exhibit defects in external ear formation
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• 71% of embryos exhibit a spectrum of inner ear defects, ranging from mild (either truncated posterior semi-circular canal or fused to the common crus) to highly abnormal (extreme inner ear bone malformation)
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• embryonic lethality between E13.5 and E15.5
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• neural tube closure defects
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• 63 % of E13.5 or older embryos exhibit exencephaly
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• kidneys show branching defects
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• short lower jaws 74% of embryos
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• reduction in bone density in the cranium, nasal cavity, ulna, humerus, mandible and femur
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• delay in bone formation in embryos
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• in 59% of embryos
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Mouse Models of Human Disease |
DO ID | OMIM ID(s) | Ref(s) | |
| CHARGE syndrome | DOID:0050834 |
OMIM:214800 |
J:217080 | |
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• no mice are found as early as E8.5
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• protected against inflammatory arthritis
• otherwise viable, fertile, and phenotypically normal
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• protected against inflammatory arthritis
• otherwise viable, fertile, and phenotypically normal
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• viable, fertile, and phenotypically normal
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
| N |
• mice are fertile
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• aggrecan loss (as measured by loss of toiludine blue staining in the tibiofemoral joint) is significantly less (1.15+/-0.15 mean score compared to 1.94+/-0.13 in wild-type joints)
• tibial cartilage erosion is seen in 1 of 13 joints as compared to 5 of 14 wild-type joints
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• mice show reduced ability to maintain balance on rotarod; performance depends on rate of rotation, such that at low speeds, mice show no deficiency
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• small number of Purkinje cells have abnormal orientation deviating from normal orientation where cell bodies are perpendicular to the surface of the cerebellar cortex
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• almost all cells show a marked decrease in branches and dendritic spines compared to wild-type
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• cell bodies of some Purkinje cells are dissociated from the cerebellar Purkinje cell layer and ectopically located in molecular layer
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• only one female in 16 live births
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• fewer female embryos from a cross of hemizygous males and homozygous cre expressing females at E13.5
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• neural tube defects of variable severity at E11.5 in females
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• in 30% of E12.5 mice
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• neural tube defects of variable severity at E11.5 in females
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• in 30% of E12.5 mice
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• exencephaly and spina bifida in 63% of mice over E12.5
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• some calvarial bones missing
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• splayed spina processes
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• in 18 of 22 mice
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• some calvarial bones missing
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• following wounding, mice show a significant reduction of epidermal melanocytes in the wound site relative to control mice
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• following wounding, mice show a significant reduction of epidermal melanocytes in the wound site relative to control mice
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• Pomc+ neurons
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• increased visceral fat mass
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• in ovariectomized mice
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• cortical dysplasia
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• hypercellularity of neuronal cells in layers 2-4
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• testis weights at 3 weeks are significantly lower (3.5 mg) than control males (24.9-27 mg)
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• testis weights at 3 weeks are significantly lower (3.5 mg) than control males (24.9-27 mg)
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• analysis of hematopoietic cells demonstrates reliable detection of YFP and RFP in same cells as well as in different cells
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• mice are born at expected Mendelian rations and do not exhibit any gross abnormalities
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• after exendin-4 treatment, mice fail to exhibit a hypophagic response unlike control mice
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• sham operated mice tended to be more glucose-intolerant than control mice
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| N |
• after vertical sleeve gastrectomy, mice exhibit normal weight changes
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• slightly fewer than expected mice with this genotype are found
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• the fraction of B and T cells expressing the recombined allele decreases during differentiation and maturation
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• fewer than expected mice with this genotype are found
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• the fraction of B and T cells expressing the recombined allele decreases during differentiation and maturation
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• mice weigh slightly less than wild type littermates at weaning and 6 months of age
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• mutants are born at a lower than expected frequency (35%)
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• only about 25% of mice survive to adulthood
• surviving mice show signs of spontaneous regression of the hyperproliferative dysregulation of erythropoiesis
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• rapid lethality in about 75% of neonatal mice
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• survivors develop intestinal inflammation similar to that in mice carrying Tg(Kit*D814V)1Roer and Tg(Mx1-cre)1Cgn
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• mice appear to die of hyperproliferative dysregulation of erythropoiesis
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• dramatic dysregulation of hematopoiesis characterized by excessive numbers of nucleated cells in the peripheral blood
• increase in nucleated cells is primarily the result of a population of small blastic cells with sparse cytoplasm
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• survivors develop mastocytosis similar to that in mice carrying Tg(Kit*D814V)1Roer and Tg(Mx1-cre)1Cgn
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• increase in the number of nuclear shadows in blood smears indicates the presence of cells with increased mechanical fragility
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• most of the liver parenchyma is replaced with Ter119+ cells
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• survivors develop intestinal inflammation similar to that in mice carrying Tg(Kit*D814V)1Roer and Tg(Mx1-cre)1Cgn
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• survivors develop mastocytosis similar to that in mice carrying Tg(Kit*D814V)1Roer and Tg(Mx1-cre)1Cgn
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
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• freshly isolated olfactory sensory neurons elicit cAMP-evoked currents with markedly reduced amplitudes
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• reduced electro-olfactogram responses to odorant containing air puffs
• olfactory bulb responses (calcium signals) are significantly reduced compared to controls
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• fail to show a preference for peanut butter odor
• fail to show an aversive reaction to 2-methyl butyric acid
• following reward training, fail to show preference for the trained scent, carvone
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• freshly isolated olfactory sensory neurons elicit cAMP-evoked currents with markedly reduced amplitudes
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• freshly isolated olfactory sensory neurons elicit cAMP-evoked currents with markedly reduced amplitudes
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
|
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
| N |
• bone marrow-derived mast cells exhibit normal ATP-stimulated citrullination
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
|
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
|
• only about 25% of mice survive to adulthood
• surviving mice show signs of spontaneous regression of the hyperproliferative dysregulation of erythropoiesis
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• rapid lethality in about 75% of neonatal mice
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• mice appear to die of hyperproliferative dysregulation of erythropoiesis
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• dramatic dysregulation of hematopoiesis characterized by excessive numbers of nucleated cells in the peripheral blood
• increase in nucleated cells is primarily the result of a population of small blastic cells with sparse cytoplasm
|
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• survivors develop mastocytosis similar to that in mice carrying Tg(Kit*D814V)3Roer and Tg(Mx1-cre)1Cgn
|
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• increase in the number of nuclear shadows in blood smears indicates the presence of cells with increased mechanical fragility
|
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• most of the liver parenchyma is replaced with Ter119+ cells
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• survivors develop mastocytosis similar to that in mice carrying Tg(Kit*D814V)3Roer and Tg(Mx1-cre)1Cgn
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
|
• homozygous embryos are present at Mendelian ratios at E10.0-E10.5 but die soon after; no viable homozygotes are recovered after E11.5
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| ♀ | phenotype observed in females |
| ♂ | phenotype observed in males |
| N | normal phenotype |
Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Models of Human Cancer database (MMHCdb) (formerly Mouse Tumor Biology (MTB)), Gene Ontology (GO) |
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last database update 12/10/2024 MGI 6.24 |
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