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Phenotypes associated with this allele
Allele Symbol
Allele Name
Allele ID 		Dcxtm1Caw
targeted mutation 1, Christopher A Walsh
MGI:2387757
Summary 15 genotypes
Jump to Allelic Composition Genetic Background Genotype ID
ht1
 		Dcxtm1Caw/Dcx+ involves: 129S6/SvEvTac * 129X1/SvJ MGI:3618280
ht2
 		Dcxtm1Caw/Dcx+ involves: 129S6/SvEvTac * 129X1/SvJ * Black Swiss MGI:3618275
cx3
 		Dclk1tm1.1Jgg/Dclk1tm1.1Jgg
Dcxtm1Caw/Dcxtm1Caw 		involves: 129S1/Sv * 129X1/SvJ * Black Swiss * C57BL/6 MGI:3618309
cx4
 		Dclk1tm1.1Jgg/Dclk1tm1.1Jgg
Dcxtm1Caw/Y 		involves: 129S1/Sv * 129X1/SvJ * Black Swiss * C57BL/6 MGI:3618307
cx5
 		Dclk1tm1.1Jgg/Dclk1+
Dcxtm1Caw/Y 		involves: 129S1/Sv * 129X1/SvJ * Black Swiss * C57BL/6 MGI:3618308
cx6
 		Dclk1tm1.1Jgg/Dclk1tm1.1Jgg
Dcxtm1Caw/Dcx+ 		involves: 129S1/Sv * 129X1/SvJ * Black Swiss * C57BL/6 MGI:3618310
cx7
 		Dclk1tm1.1Jgg/Dclk1+
Dcxtm1Caw/Dcxtm1Caw 		involves: 129S1/Sv * 129X1/SvJ * Black Swiss * C57BL/6 MGI:3618311
cx8
 		Dclk1tm1Caw/Dclk1tm1Caw
Dcxtm1Caw/Y 		involves: 129S6/SvEvTac * 129X1/SvJ * C57BL/6J MGI:3628582
cx9
 		Dclk1tm1Caw/Dclk1tm1Caw
Dcxtm1Caw/Dcx+ 		involves: 129S6/SvEvTac * 129X1/SvJ * C57BL/6J MGI:3628584
cx10
 		Dclk1tm1Caw/Dclk1+
Dcxtm1Caw/Dcx+ 		involves: 129S6/SvEvTac * 129X1/SvJ * C57BL/6J MGI:3628593
cx11
 		Dclk1tm1Caw/Dclk1+
Dcxtm1Caw/Y 		involves: 129S6/SvEvTac * 129X1/SvJ * C57BL/6J MGI:3628602
cx12
 		Dclk2tm1.2Jgg/Dclk2tm1.2Jgg
Dcxtm1Caw/Dcxtm1Caw 		involves: 129/Sv * C57BL/6 MGI:3844814
cx13
 		Dclk2tm1.2Jgg/Dclk2tm1.2Jgg
Dcxtm1Caw/Y 		involves: 129/Sv * C57BL/6 MGI:3844816
ot14
 		Dcxtm1Caw/Y involves: 129S6/SvEvTac * 129X1/SvJ * Black Swiss MGI:3618273
ot15
 		Dcxtm1Caw/Y involves: 129/Sv * C57BL/6 MGI:3844817


Genotype
MGI:3618280
ht1
Allelic
Composition		 Dcxtm1Caw/Dcx+
Genetic
Background		 involves: 129S6/SvEvTac * 129X1/SvJ
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
behavior/neurological
abnormal avoidance learning behavior ( J:78746 )
• female mice show impaired ability to associate stimulus (tone) with unconditioned stimulus (shock) in the fear test
abnormal spatial learning ( J:78746 )
• animals demonstrate mild deficits in latency to find hidden platform in Morris water maze test
decreased grip strength ( J:78746 )
• female heterozygous mice show deficits in cage-top hang tests




Genotype
MGI:3618275
ht2
Allelic
Composition		 Dcxtm1Caw/Dcx+
Genetic
Background		 involves: 129S6/SvEvTac * 129X1/SvJ * Black Swiss
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
nervous system
abnormal hippocampus morphology ( J:78746 )
• hippocampal formation is malformed
abnormal hippocampus pyramidal cell layer ( J:78746 )
• pyramidal layer of CA3 is split in some regions
• pyramidal neurons are more loosely arrayed in some animals
• milder dispruptions of the pyramidal layerare seen in CA2 and CA1 regions

behavior/neurological
abnormal avoidance learning behavior ( J:78746 )
• female mice showed impaired ability to associate stimulus (tone) with unconditioned stimulus (shock) in a fear test
decreased grip strength ( J:78746 )
• female mice show deficits in cage-top hang tests




Genotype
MGI:3618309
cx3
Allelic
Composition		 Dclk1tm1.1Jgg/Dclk1tm1.1Jgg
Dcxtm1Caw/Dcxtm1Caw
Genetic
Background		 involves: 129S1/Sv * 129X1/SvJ * Black Swiss * C57BL/6
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk1tm1.1Jgg mutation (0 available); any Dclk1 mutation (55 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
mortality/aging
postnatal lethality, complete penetrance ( J:105300 )
• pups die before weaning, usually soon after birth

nervous system
absent corpus callosum ( J:105300 )
• corpus callosum is absent
abnormal stratification in cerebral cortex ( J:105300 )
• there is no apparent laminar structure to the cortex
abnormal neuron morphology ( J:105300 )
• cortical neuron distribution is altered, showing a broader pattern of distribution




Genotype
MGI:3618307
cx4
Allelic
Composition		 Dclk1tm1.1Jgg/Dclk1tm1.1Jgg
Dcxtm1Caw/Y
Genetic
Background		 involves: 129S1/Sv * 129X1/SvJ * Black Swiss * C57BL/6
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk1tm1.1Jgg mutation (0 available); any Dclk1 mutation (55 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
mortality/aging
postnatal lethality, complete penetrance ( J:105300 )
• pups die before weaning, usually shortly after birth

nervous system
absent corpus callosum ( J:105300 )
• corpus callosum is absent
abnormal anterior commissure morphology ( J:105300 )
• anterior commissure is hypoplastic
abnormal stratification in cerebral cortex ( J:105300 )
• a cortical lamination defect, most notable in the lateral cortex is apparent
• cytoarchitectonics are disrupted; there is dispersion of the cell-rich layer 2/3




Genotype
MGI:3618308
cx5
Allelic
Composition		 Dclk1tm1.1Jgg/Dclk1+
Dcxtm1Caw/Y
Genetic
Background		 involves: 129S1/Sv * 129X1/SvJ * Black Swiss * C57BL/6
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk1tm1.1Jgg mutation (0 available); any Dclk1 mutation (55 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
nervous system
abnormal corpus callosum morphology ( J:105300 )
• corpus callosum is hypoplastic
decreased anterior commissure size ( J:105300 )
• anterior commissure is severely reduced in thickness
abnormal neuron morphology ( J:105300 )
• cortical neuron distribution is altered, with neurons showing a much broader pattern of distribution




Genotype
MGI:3618310
cx6
Allelic
Composition		 Dclk1tm1.1Jgg/Dclk1tm1.1Jgg
Dcxtm1Caw/Dcx+
Genetic
Background		 involves: 129S1/Sv * 129X1/SvJ * Black Swiss * C57BL/6
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk1tm1.1Jgg mutation (0 available); any Dclk1 mutation (55 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
nervous system
absent corpus callosum ( J:105300 )
• corpus callosum is absent
abnormal anterior commissure morphology ( J:105300 )
• anterior commissure is hypoplastic
abnormal stratification in cerebral cortex ( J:105300 )
• cytoarchitectonics are disrupted; there is dispersion of the cell-rich layer 2/3




Genotype
MGI:3618311
cx7
Allelic
Composition		 Dclk1tm1.1Jgg/Dclk1+
Dcxtm1Caw/Dcxtm1Caw
Genetic
Background		 involves: 129S1/Sv * 129X1/SvJ * Black Swiss * C57BL/6
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk1tm1.1Jgg mutation (0 available); any Dclk1 mutation (55 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
nervous system
absent corpus callosum ( J:105300 )
• corpus callosum is absent




Genotype
MGI:3628582
cx8
Allelic
Composition		 Dclk1tm1Caw/Dclk1tm1Caw
Dcxtm1Caw/Y
Genetic
Background		 involves: 129S6/SvEvTac * 129X1/SvJ * C57BL/6J
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk1tm1Caw mutation (0 available); any Dclk1 mutation (55 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
mortality/aging
postnatal lethality, incomplete penetrance ( J:105301 )
• more than 75% of double knockouts die within 2 days of birth

nervous system
abnormal brain morphology ( J:105301 )
• double knockouts exhibit disruption of most or all major axon tracts in the brain; brains of double knockouts display severely disorganized white-matter tracts
abnormal cerebellar foliation ( J:105301 )
• cerebellum of mutants has defective foliation; folia are severely hypoplastic, particularly on the ventral surface of the cerebellum
abnormal corpus callosum morphology ( J:105301 )
• there are no fibers crossing the midline of the corpus callosum; most axons end before reaching the midline and do not cross to the other side
• axons appear as multiple single axons with random orientation and without clear fasciculation rather than bundles
decreased corpus callosum size ( J:105301 )
• in mutants, corpus callosum is present at rostral levels but does not extend as far caudally as in wild-type mice; anterior portion is greatly reduced in thickness
abnormal hippocampal commissure morphology ( J:105301 )
• hippocampal commissures appear lumpy and disorganized
abnormal anterior commissure morphology ( J:105301 )
• aberrant anterior commissure bundles are observed
abnormal brain internal capsule morphology ( J:105301 )
• in some animals, internal capsule has abnormal morphology
absent brain internal capsule ( J:105301 )
• there is absence of an internal capsule in the striatum
abnormal brainstem morphology ( J:105301 )
• disruption of many brainstem nuclei is observed
abnormal striatum morphology ( J:105301 )
• there is absence of pencil fibers in the striatum
abnormal thalamus morphology ( J:105301 )
• disruption of many thalamic nuclei is observed
abnormal cerebral cortex morphology ( J:105301 )
• stratum pyramidale layer in pyramidal cell fields, CA3,CA2and CA1 is disorganized with multiple cell layers separated by irregular streams of white matter
abnormal stratification in cerebral cortex ( J:105301 )
• at P0, there is disruption of lamination of the cerebral cortex in the cingulate gyrus in double knockouts
thin cerebral cortex ( J:105301 )
• brains of double knockouts at P0 display a thin cerebral cortex
abnormal entorhinal cortex morphology ( J:105301 )
• no axons leave the region of the entorhinal cortex
abnormal hippocampus morphology ( J:105301 )
• subcortical white matter appears lumpy and disorganized
abnormal dentate gyrus morphology ( J:105301 )
• cells appear less tightly packed
abnormal hippocampus layer morphology ( J:105301 )
• there is markedly abnormal cell layering in the hippocampus
small cerebellum ( J:105301 )
• the cerebellum is small in double knockouts




Genotype
MGI:3628584
cx9
Allelic
Composition		 Dclk1tm1Caw/Dclk1tm1Caw
Dcxtm1Caw/Dcx+
Genetic
Background		 involves: 129S6/SvEvTac * 129X1/SvJ * C57BL/6J
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk1tm1Caw mutation (0 available); any Dclk1 mutation (55 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
reproductive system
reduced female fertility ( J:105301 )
• fertility appears to be reduced




Genotype
MGI:3628593
cx10
Allelic
Composition		 Dclk1tm1Caw/Dclk1+
Dcxtm1Caw/Dcx+
Genetic
Background		 involves: 129S6/SvEvTac * 129X1/SvJ * C57BL/6J
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk1tm1Caw mutation (0 available); any Dclk1 mutation (55 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
reproductive system
reduced female fertility ( J:105301 )
• fertility appears to be reduced




Genotype
MGI:3628602
cx11
Allelic
Composition		 Dclk1tm1Caw/Dclk1+
Dcxtm1Caw/Y
Genetic
Background		 involves: 129S6/SvEvTac * 129X1/SvJ * C57BL/6J
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk1tm1Caw mutation (0 available); any Dclk1 mutation (55 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
mortality/aging
postnatal lethality ( J:105301 )
• a slightly lower proportion of Dcx-deficient, Dcamkl1-heterozygous males die postnatally than double knockouts

nervous system
abnormal brain morphology ( J:105301 )
• mutants have less severe and less penetrant white matter defects than double knockouts
enlarged lateral ventricles ( J:105301 )
• lateral ventricles are enlarged in mutants
absent corpus callosum ( J:105301 )
• some animals have an absent corpus callosum




Genotype
MGI:3844814
cx12
Allelic
Composition		 Dclk2tm1.2Jgg/Dclk2tm1.2Jgg
Dcxtm1Caw/Dcxtm1Caw
Genetic
Background		 involves: 129/Sv * C57BL/6
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk2tm1.2Jgg mutation (1 available); any Dclk2 mutation (135 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
mortality/aging
premature death ( J:148345 )
• less than 10% of mice survive to 5 months of age
postnatal lethality, incomplete penetrance ( J:148345 )
• about half the die mice before weaning

behavior/neurological
convulsive seizures ( J:148345 )
• repetitive epileptiform discharges from the hippocampus consisting of 4-Hz monomorphic spike activity are observed in mice prior to seizures
• at the onset of the convulsive attack, hippocampus activity increases from 400 microV up to 1 mV, and then transitions into spike and wave appearance at 3-Hz frequency
• these spikes in activity transmit to the cortex about half the time leading to tonic-clonic convulsions
tonic-clonic seizures ( J:148345 )
• as early as P16, mice have spontaneous seizures often accompanied by behavioral arrest and forelimb myoclonus
• seizures originate in the hippocampus with spikes in activity that half the time transmit to the cortex and cause tonic-clonic seizures

nervous system
convulsive seizures ( J:148345 )
• repetitive epileptiform discharges from the hippocampus consisting of 4-Hz monomorphic spike activity are observed in mice prior to seizures
• at the onset of the convulsive attack, hippocampus activity increases from 400 microV up to 1 mV, and then transitions into spike and wave appearance at 3-Hz frequency
• these spikes in activity transmit to the cortex about half the time leading to tonic-clonic convulsions
tonic-clonic seizures ( J:148345 )
• as early as P16, mice have spontaneous seizures often accompanied by behavioral arrest and forelimb myoclonus
• seizures originate in the hippocampus with spikes in activity that half the time transmit to the cortex and cause tonic-clonic seizures
abnormal brain interneuron morphology ( J:148345 )
• CaBP+ interneurons that interact with pyramidal neurons are improperly laminated within the stratum radiatum-lacunosum moleculare border
abnormal dentate gyrus morphology ( J:148345 )
• mice have reduced packing density of the dentate granule neuron layer, resulting in an increased thickness
abnormal hippocampal mossy fiber morphology ( J:148345 )
• mossy fibers fail to separate into the infrapyramidal and the suprapyrimidal bundles and, as a result, mossy fibers travel aberrantly within the pyramidal layer
abnormal hippocampus granule cell layer ( J:148345 )
• lamination density of the granule cell layer is increased in these mice
abnormal hippocampus pyramidal cell morphology ( J:148345 )
• pyramidal neurons displayed a striking simplification of apical dendritic arbors with decreased branching from both primary and secondary apical dendrites
abnormal hippocampus region morphology ( J:148345 )
• about half of the somatostatin neurons are selectively lost in the hippocampus by day 18 after birth
abnormal hippocampus CA1 region morphology ( J:148345 )
• about 40% of neurons are displaced into the stratum oriens
abnormal hippocampus CA3 region morphology ( J:148345 )
• the CA3 region is dyslaminated with a small percentage of neurons heterotopically located in the stratum oriens
abnormal inhibitory postsynaptic currents ( J:148345 )
• the frequency of spontaneous sIPSC among pyramidal neurons is severely reduced by 55% compared to controls




Genotype
MGI:3844816
cx13
Allelic
Composition		 Dclk2tm1.2Jgg/Dclk2tm1.2Jgg
Dcxtm1Caw/Y
Genetic
Background		 involves: 129/Sv * C57BL/6
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dclk2tm1.2Jgg mutation (1 available); any Dclk2 mutation (135 available)
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype

Hippocampal disorganization, increased c-fos, and loss of SOM+ interneurons in Dcxtm1Caw/Y Dclk2tm1.2Jgg/Dclk2tm1.2Jgg mice

mortality/aging
premature death ( J:148345 )
• less than 10% of mice survive to 5 months of age
postnatal lethality, incomplete penetrance ( J:148345 )
• about half the die mice before weaning

behavior/neurological
convulsive seizures ( J:148345 )
• repetitive epileptiform discharges from the hippocampus consisting of 4-Hz monomorphic spike activity are observed in mice prior to seizures
• at the onset of the convulsive attack, hippocampus activity increases from 400 microV up to 1 mV, and then transitions into spike and wave appearance at 3-Hz frequency
• these spikes in activity transmit to the cortex about half the time leading to tonic-clonic convulsions
tonic-clonic seizures ( J:148345 )
• as early as P16, mice have spontaneous seizures often accompanied by behavioral arrest and forelimb myoclonus
• seizures originate in the hippocampus with spikes in activity that half the time transmit to the cortex and cause tonic-clonic seizures

nervous system
convulsive seizures ( J:148345 )
• repetitive epileptiform discharges from the hippocampus consisting of 4-Hz monomorphic spike activity are observed in mice prior to seizures
• at the onset of the convulsive attack, hippocampus activity increases from 400 microV up to 1 mV, and then transitions into spike and wave appearance at 3-Hz frequency
• these spikes in activity transmit to the cortex about half the time leading to tonic-clonic convulsions
tonic-clonic seizures ( J:148345 )
• as early as P16, mice have spontaneous seizures often accompanied by behavioral arrest and forelimb myoclonus
• seizures originate in the hippocampus with spikes in activity that half the time transmit to the cortex and cause tonic-clonic seizures
abnormal brain interneuron morphology ( J:148345 )
• CaBP+ interneurons that interact with pyramidal neurons are improperly laminated within the stratum radiatum-lacunosum moleculare border
abnormal dentate gyrus morphology ( J:148345 )
• mice have reduced packing density of the dentate granule neuron layer, resulting in an increased thickness
abnormal hippocampal mossy fiber morphology ( J:148345 )
• mossy fibers fail to separate into the infrapyramidal and the suprapyrimidal bundles and, as a result, mossy fibers travel aberrantly within the pyramidal layer
abnormal hippocampus granule cell layer ( J:148345 )
• lamination density of the granule cell layer is increased in these mice
abnormal hippocampus pyramidal cell morphology ( J:148345 )
• pyramidal neurons displayed a striking simplification of apical dendritic arbors with decreased branching from both primary and secondary apical dendrites
abnormal hippocampus region morphology ( J:148345 )
• about half of the somatostatin neurons are selectively lost in the hippocampus by day 18 after birth
abnormal hippocampus CA1 region morphology ( J:148345 )
• about 40% of neurons are displaced into the stratum oriens
abnormal hippocampus CA3 region morphology ( J:148345 )
• the CA3 region is dyslaminated with a small percentage of neurons heterotopically located in the stratum oriens
abnormal inhibitory postsynaptic currents ( J:148345 )
• the frequency of spontaneous sIPSC among pyramidal neurons is severely reduced by 55% compared to controls




Genotype
MGI:3618273
ot14
Allelic
Composition		 Dcxtm1Caw/Y
Genetic
Background		 involves: 129S6/SvEvTac * 129X1/SvJ * Black Swiss
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype
mortality/aging
premature death ( J:78746 )
• only a small number of males survive to adulthood
postnatal lethality, incomplete penetrance ( J:78746 )
• majority of hemizygous males die within a few days of birth

growth/size/body
decreased body size ( J:78746 )
• surviving males are runted compared to littermates

behavior/neurological
abnormal behavior ( J:78746 )
• hemizygous male mice tested show similar deficits to heterozygous female littermates in behavioral tests

nervous system
abnormal hippocampus morphology ( J:78746 )
• hippocampal formation is malformed
abnormal hippocampus pyramidal cell layer ( J:78746 )
• pyramidal layer of CA3 is split in some regions
• pyramidal neurons are more loosely arrayed in some animals
• there are milder disruptions of the pyramidal layer in CA2 And CA1 regions

reproductive system
reduced male fertility ( J:78746 )
• males show variable fertility




Genotype
MGI:3844817
ot15
Allelic
Composition		 Dcxtm1Caw/Y
Genetic
Background		 involves: 129/Sv * C57BL/6
Find Mice Using the International Mouse Strain Resource (IMSR)
Mouse lines carrying:
Dcxtm1Caw mutation (0 available); any Dcx mutation (20 available)
♀ phenotype observed in females
♂ phenotype observed in males
N normal phenotype

Dyslaminated interneuron populations in Dcxtm1Caw/Y Dclk2tm1.2Jgg/Dclk2tm1.2Jgg and Dcxtm1Caw/Y mice

nervous system
abnormal hippocampus CA3 region morphology ( J:148345 )
• the CA3 region is dyslaminated by 11 days of age
abnormal hippocampal mossy fiber morphology ( J:148345 )
• the infrapyramidal mossy fiber bundle crosses the pyramidal layer and merges prematurely with the suprapyrimidal bundle
abnormal inhibitory postsynaptic currents ( J:148345 )
• the frequency of spontaneous sIPSC among pyramidal neurons is somewhat reduced compared to controls




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Send questions and comments to User Support. 		last database update
11/12/2024
MGI 6.24 		The Jackson Laboratory