Analysis Tools
immune system
| N |
• mice exhibit normal B cell development and T cell-independent immunological response
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• 2.5-fold in mice treated with sheep red blood cells or infected with N. b.
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• mice infected with N. b. exhibit a 1.6-fold increased in IgG1 and 2.9-fold increase in IgE plasma cells compared with wild-type mice
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• in mice treated with sheep red blood cells
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• mice infected with N. b. exhibit enhanced primary and secondary IgE response compared with wild-type mice
• mice infected with N. b. exhibit increased IgE-expressing cells in the extrafollicular regions of the spleen compared with wild-type mice
• increased IgE-expressing cells in the extrafollicular regions of the spleen and mesenteric lymph nodes in mice treated with sheep red blood cells
• mice infected with N. b. exhibit a 1.6-fold increased in IgG1 and 2.9-fold increase in IgE plasma cells compared with wild-type mice
• however, mice exhibit normal IgE-expressing germinal center cells in mesenteric lymph nodes in N. b. infected mice
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• mice treated with sheep red blood cells exhibit increased IgG1 response compared with wild-type mice
• mice infected with N. b. exhibit enhanced numbers of IgG1high cells in mesenteric lymph nodes compared with wild-type mice
• mice infected with N. b. exhibit a 1.6-fold increased in IgG1 and 2.9-fold increase in IgE plasma cells compared with wild-type mice
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• mice treated with sheep red blood cells exhibit increased IgG2a response compared with wild-type mice
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• mice infected with N. b. exhibit enhanced primary and secondary IgM response compared with wild-type mice
• mice infected with N. b. exhibit increased IgM-expressing extrafollicular plasmablasts in the spleen compared with wild-type mice
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• mice treated with Nippostrongylus brasiliensis (N. b.) infection exhibit enhanced primary and secondary IgE and IgM responses compared with wild-type mice
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nervous system
| N |
• primary neurons exhibit normal endocytosis and exocytosis
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• slow axonal transport
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hematopoietic system
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• 2.5-fold in mice treated with sheep red blood cells or infected with N. b.
|
|
• mice infected with N. b. exhibit a 1.6-fold increased in IgG1 and 2.9-fold increase in IgE plasma cells compared with wild-type mice
|
|
• in mice treated with sheep red blood cells
|
|
• mice infected with N. b. exhibit enhanced primary and secondary IgE response compared with wild-type mice
• mice infected with N. b. exhibit increased IgE-expressing cells in the extrafollicular regions of the spleen compared with wild-type mice
• increased IgE-expressing cells in the extrafollicular regions of the spleen and mesenteric lymph nodes in mice treated with sheep red blood cells
• mice infected with N. b. exhibit a 1.6-fold increased in IgG1 and 2.9-fold increase in IgE plasma cells compared with wild-type mice
• however, mice exhibit normal IgE-expressing germinal center cells in mesenteric lymph nodes in N. b. infected mice
|
|
• mice treated with sheep red blood cells exhibit increased IgG1 response compared with wild-type mice
• mice infected with N. b. exhibit enhanced numbers of IgG1high cells in mesenteric lymph nodes compared with wild-type mice
• mice infected with N. b. exhibit a 1.6-fold increased in IgG1 and 2.9-fold increase in IgE plasma cells compared with wild-type mice
|
|
• mice treated with sheep red blood cells exhibit increased IgG2a response compared with wild-type mice
|
|
• mice infected with N. b. exhibit enhanced primary and secondary IgM response compared with wild-type mice
• mice infected with N. b. exhibit increased IgM-expressing extrafollicular plasmablasts in the spleen compared with wild-type mice
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