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| Caption | Keratinocyte-specific deletion of Smad2 results in accelerated re-epithelialization during wound healing. B, wound widths were determined by K14 (brown) immunostaining in Tg(KRT5-cre)1Xya/0 Smad2tm1Xya/Smad2tm1Xya (K5-Cre;Smad2fl/fl) and control (K5-Cre;Smad2fl/+) wounded skin at days 3, 5, and 7 after injury. Arrows indicated the leading edges of wounded epidermis. | ||||||
| Copyright | This image is from Yang X, J Biol Chem 2011 Dec 9;286(49):42267-73 and is displayed with the permission of the American Society for Biochemistry and Molecular Biology who owns the Copyright. Full text from JBC. J:178733 | ||||||
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Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Models of Human Cancer database (MMHCdb) (formerly Mouse Tumor Biology (MTB)), Gene Ontology (GO) |
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last database update 12/10/2024 MGI 6.24 |
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