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Mapping Data
Experiment
  • Experiment
    TEXT-QTL
  • Chromosome
    13
  • Reference
    J:53193 Butterfield RJ, et al., Genetic analysis of disease subtypes and sexual dimorphisms in mouse experimental allergic encephalomyelitis (EAE): relapsing/remitting and monophasic remitting/nonrelapsing EAE are immunogenetically distinct. J Immunol. 1999 Mar 1;162(5):3096-102
  • ID
    MGI:1342794
Genes
GeneAlleleAssay TypeDescription
Eae13 visible phenotype
D13Mit66
Notes
  • Experiment
    Analysis of 633 (SJL/J x B10.S/DvTe)F2 mice with induced experimental allergic encephalomyelitis (EAE) indicated the presence of a quantitative locus (QTL) involved with the monophasic remitting/nonrelapsing (M-RNR) form of the disorder. Eae13, showed maximum linkage to D13Mit66 at approximately 37 cM on mouse Chromosome 13. Stratification of the data by sex indicated that the effect of Eae13 was greater in males that in females. Susceptibility is associated with the SJL allele.

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Mouse Genome Database (MGD), Gene Expression Database (GXD), Mouse Models of Human Cancer database (MMHCdb) (formerly Mouse Tumor Biology (MTB)), Gene Ontology (GO)
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last database update
11/12/2024
MGI 6.24
The Jackson Laboratory